Editing Chest standard texts (section)

== COVID-19 - in progress ==

{| class="wikitable mw-collapsible <!--mw-collapsed-->"
|-
||''Text to copy and insert, optimised for Sectra standard report texts.&nbsp;''
|-
|
'''Auto-inserted clinical details:'''<br />
<RequestCaseHistory><br />
<RequestReason><br />

'''Report:'''<br />
Scan quality: [Good/Adequate/Poor/Non-diagnostic.].<br />
Peripheral, bilateral GGO with or without
consolidation and with or without intralobular
lines (crazy paving)? [Yes/No.]<br />
Location: [Saddle/Main left/Main right/Segmental left/Segmental right/Subsegmental left/Subsegmental right/Multiple/Right heart]<br />
Features of right heart strain/raised pulmonary artery pressure: [Straightening/reverse bowing of interventricular septum/Pulmonary artery enlargement/Reflux of contrast into hepatic veins]<br />
Right ventricular/Left ventricular diameter ratio: []<br />
Lymphadenopathy: [Axillary/Hilar/Mediastinal]<br />
Lung parenchyma: [] <br />
Pleural disease: [] <br /> 
Pulmonary nodules: [] <br /> 
Upper abdominal organs: [] <br /> 
Bones: [] <br /> 
Conclusion: [Commonly reported imaging features of classic COVID-19 pneumonia are present/Imaging findings present are nonspecific, but can be seen in COVID-19 pneumonia/Imaging findings are not commonly reported in COVID-19 pneumonia. Alternative diagnoses should be considered] <br /> 
|}
{| class="wikitable mw-collapsible <!--mw-collapsed-->"
|colspan="3"| Explanatory notes&nbsp;
|-
!Heading
!Options
!Reason
|-
||Pulmonary emboli? ||Yes/No. ||
|-
||Location ||Saddle/Main left/Main right/Segmental left/Segmental right/Subsegmental left/Subsegmental right/Multiple/Right heart ||High clot burden may correlate with prognosis (not all studies find this to be so) and the PE index (Wu et al) may predict outcome, but the most significant factor is whether there is haemodynamic compromise/shock. Right heart clot is associated with increased mortality.
|-
||Features of right heart strain/raised pulmonary artery pressure ||Straightening/reverse bowing of interventricular septum/Pulmonary artery enlargement/Reflux of contrast into hepatic veins ||Classification of PE into massive, submassive and nonmassive (high, intermediate and low risk) does not rely on CT and relies on the patient's clincal status specifically blood pressure and other signs of shock. However, various combinations of CT and clinical signs have been studied and shown to predict mortality - Jaff et al provide a good summary. 
|-
||Right ventricular/Left ventricular diameter ratio: || ||Originally assessed on the 4 chamber view, but you may not need to reformat - measurements on axial images (not necessarily the same images) have been shown to give equivalent results. > 0.9 is significant, although not across all studies - reformatting into a 4 chamber view is quick and easy. Again various combinations of this ratio and other factors may help stratify patients e.g. Kyoung Kang et al. Give measurements and ratio e.g. RV/LV = 45 mm/40 mm = 1.125
|-
||Lymphadenopathy: ||Axillary/Hilar/Mediastinal 
|rowspan="4"|Many other features are likely to contribute to prognosis including acute infection and cancer.
|-
||Lung parenchyma: || 

|-
||Pleural disease: ||  
|-
||Pulmonary nodules: || 
|-
||Upper abdominal organs: || ||
|-
||Bones || ||
|-
||Vasculature including coronary arteries || ||High pitch CTPAs will often demonstrate some detail relating to the coronary arteries.
|}

''References:''
1. <br />